Given their pivotal role in apoptosis, caspases are attractive targets for therapeutic intervention. Inhibitors of caspases are being explored for treating conditions where excessive apoptosis is detrimental, such as stroke or myocardial infarction. Conversely, caspase activation is being investigated as a strategy to induce apoptosis in cancer cells. However, the challenge lies in achieving specificity, as caspases are involved in multiple pathways.
